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myocardium and posterior
As the left posterior fascicle is shorter and broader than the right, impulses reach the ventricular muscles just prior to depolarization, and therefore contraction, of the left ventricle myocardium.

myocardium and left
Due to the hypertrophy of the left ventricle in aortic stenosis, including the consequent inability of the coronary arteries to adequately supply blood to the myocardium ( see " Angina " below ), arrhythmias may develop.
During left ventricular diastole, after the pressure drops in the left ventricle due to relaxation of the ventricular myocardium, the mitral valve opens, and blood travels from the left atrium to the left ventricle.
This early filling phase is due to active relaxation of the ventricular myocardium, causing a pressure gradient that allows a rapid flow of blood from the left atrium, across the mitral valve.
Triggered by Electrocardiogram ( EKG ) to obtain differential information about the heart in various parts of its cycle, gated myocardial SPECT can be used to obtain quantitative information about myocardial perfusion, thickness, and contractility of the myocardium during various parts of the cardiac cycle, and also to allow calculation of left ventricular ejection fraction, stroke volume, and cardiac output.
After a brief delay at the AV node, the stimulus is conducted through the bundle of His to the left and right bundle branches and then to the Purkinje fibers and the endocardium at the apex of the heart, then finally to the ventricular myocardium.
During the ventricular contraction portion of the cardiac cycle, the Purkinje fibers carry the contraction impulse from both the left and right bundle branch to the myocardium of the ventricles.
Rheumatic heart disease at autopsy with characteristic findings ( thickened mitral valve, thickened chordae tendineae, hypertrophied left ventricular myocardium ).
The resulting ischemia ( restriction in blood supply ) and oxygen shortage, if left untreated for a sufficient period of time, can cause damage or death of heart muscle tissue ( myocardium ).
Imaging features of restrictive cardiomyopathy demonstrate an increased left ventricular thickness with infiltration of the myocardium.
Rheumatic heart disease at autopsy with characteristic findings ( thickened mitral valve, thickened chordae tendineae, hypertrophied left ventricular myocardium ).
This electrical impulse is propagated throughout the right atrium, and through Bachmann's bundle to the left atrium, stimulating the myocardium of both atria to contract.
In this phase, the ventricular myocardium is no longer able to contract adequately to compensate for the volume overload of mitral regurgitation, and the stroke volume of the left ventricle will decrease.
Left ventricular hypertrophy ( LVH ) is the thickening of the myocardium ( muscle ) of the left ventricle of the heart.
Normal thickness of the left ventricular myocardium is from 0. 6 to 1. 1 cm ( as measured at the very end of diastole.
These impulses are then able to travel through the myocardium of the left ventricle to the right ventricle and depolarise the right ventricle this way.
After the insult occurs, a series of histopathological and structural changes occur in the left ventricular myocardium that lead to progressive decline in left ventricular performance.
Atrial systole represents the contraction of myocardium of the left and right atria.
Ventricular systole is a written description of the contraction of the myocardium of the left and right ventricles.

myocardium and ventricle
It is characterized by hypokinetic areas involving the free wall of the right ventricle, with fibrofatty replacement of the right ventricular myocardium, with associated arrhythmias originating in the right ventricle.
Kussmaul's sign suggests impaired filling of the right ventricle due to either fluid in the pericardial space or a poorly compliant myocardium or pericardium.

myocardium and replaced
After myocardial infarction, dead myocytes are replaced by scar tissue, deleteriously affecting the function of the myocardium.

myocardium and by
:::; Takotsubo cardiomyopathy ( Transient apical ballooning, stress-induced cardiomyopathy ): A type of dilated cardiomyopathy caused by a sudden temporary weakening of the myocardium.
The larvae travel by capillaries to various organs, such as the retina, myocardium, or lymph nodes ; however, only larvae that migrate to skeletal muscle cells survive and encyst.
If one coronary artery is obstructed by an atheroma, the second artery is still able to supply oxygenated blood to the myocardium.
This is usually by application of an electric shock to the myocardium and is discussed in detail in the relevant article.
Heart failure is caused by any condition which reduces the efficiency of the myocardium, or heart muscle, through damage or overloading.
Initially, this helps compensate for heart failure by maintaining blood pressure and perfusion, but places further strain on the myocardium, increasing coronary perfusion requirements, which can lead to worsening of ischemic heart disease.
* Hypertrophy ( an increase in physical size ) of the myocardium, caused by the terminally differentiated heart muscle fibres increasing in size in an attempt to improve contractility.
Many secondary causes, such as a heart attack, can lead to inflammation of the myocardium and therefore the diagnosis of myocarditis cannot be made by evidence of inflammation of the myocardium alone.
A small tissue sample of the endocardium and myocardium is taken, and investigated by a pathologist by light microscopy and — if necessary — immunochemistry and special staining methods.
Cardiac magnetic resonance imaging ( cMRI or CMR ) has been shown to be very useful in diagnosing myocarditis by visualizing markers for inflammation of the myocardium.
In the atria the electrical signal moves from cell to cell while in the ventricles the signal is carried by specialized tissue called the Purkinje fibers which then transmit the electric charge to the myocardium.
In vivo sympathetic outflow within the myocardium is probably best described by the time honored description of the sinoatrial tree branching out to Purkinges fibers.
Parasympathetic inflow within the myocardium is probably best described by influence of the vagus nerve and spinal accessory ganglia.
In another instance, a young man developed cardiac abnormalities similar to those seen in Takotsubo cardiomyopathy, characterized by a sudden temporary weakening of the myocardium.
ARVD is caused by genetic defects of the parts of heart muscle ( also called myocardium or cardiac muscle ) known as desmosomes, areas on the surface of heart muscle cells which link the cells together.
Although in many cases no cause ( etiology ) is apparent, dilated cardiomyopathy is probably the result of damage to the myocardium produced by a variety of toxic, metabolic, or infectious agents.
The total time taken by the nerve impulse to travel from the SA node to the ventricular myocardium is 0. 19 seconds.
The increase in stroke volume is explained by the Frank – Starling mechanism, in which increased ventricular pre-load stretches the myocardium such that contractions are more forceful.
After the AV canal boundary formation, a subset of endocardial cells lining the AV canal are activated by signals emanating from the myocardium and by interendocardial signaling pathways to undergo EMT.

0.118 seconds.