** Endometrial epithelial cell ( see endometrium )

** Tumours of ovarian epithelial types

** Caseinates, sodium or calcium salts of casein

** voltage-gated sodium channels NaVs

** Salt ( sodium ) and water retention

** SDS-PAGE, sodium dodecyl sulfate polyacrylamide gel electrophoresis

** Phosphorylation of Na < sup >+</ sup >/ K < sup >+</ sup >- ATPase during the transport of sodium ( Na < sup >+</ sup >) and potassium ( K < sup >+</ sup >) ions across the cell membrane in osmoregulation to maintain homeostasis of the body's water content.

** E627 Disodium guanylate, sodium guanylate

** E640 Glycine and its sodium salt

** Nitratine, the mineral form of sodium nitrate

** Often sodium silicate, this prevents corrosion of dishwasher components.

** 2 – 21 mmol / L sodium ( lower than blood plasma )

** increased salt intake also can increase body fluid volume as well as increasing urine sodium excretion, which further increases urinary calcium excretion ( In other words, calcium and sodium ( salt ) are handled in a similar way by the kidney.

** Singulair ( montelukast sodium )

** Glauber's salt, sodium sulfate

** Creatinine, urea / blood urea nitrogen, sodium, potassium, chloride, bicarbonate, calcium, magnesium, phosphate

** barbiturates such as phenobarbital, sodium thiopental and secobarbital

** i = 1. 9 for sodium chloride in water, due to the near full dissociation of NaCl into Na < sup >+</ sup > and Cl < sup >-</ sup > ( often simplified as 2 )

** SDS-PAGE, sodium dodecyl sulfate polyacrylamide gel electrophoresis, commonly used to analyse proteins

** Cation channels of sperm: This small family of channels, normally referred to as Catsper channels, is related to the two-pore channels and distantly related to TRP channels.

** Inward-rectifier potassium channels: These channels allow potassium to flow into the cell in an inwardly rectifying manner, i. e., potassium flows effectively into, but not out of, the cell.

** Calcium-activated potassium channels: This family of channels is, for the most part, activated by intracellular Ca < sup > 2 +</ sup > and contains 8 members.

** Two-pore-domain potassium channels: This family of 15 members form what is known as leak channels, and they follow Goldman-Hodgkin-Katz ( open ) rectification.

** Cyclic nucleotide-gated channels: This family of channels is characterized by activation due to the binding of intracellular cAMP or cGMP, with specificity varying by member.

** Hyperpolarization-activated cyclic nucleotide-gated channels

** Voltage-gated potassium channels e. g., Kvs, Kirs etc.

** Calcium-activated potassium channels e. g., BKCa or MaxiK, SK, etc.

** Inward-rectifier potassium channels

** Two-pore-domain potassium channels: This family of 15 members form what is known as leak channels, and they follow Goldman-Hodgkin-Katz ( open ) rectification.

** Voltage-gated proton channels

** Most Transient receptor potential channels

** Freeview digital free satellite with a dozen SD channels, with SD feeds of the terrestrial HD freeview channels.

* epithelial sodium channel

CFTR regulates the movement of chloride and sodium ions across epithelial membranes, such as the alveolar epithelia located in the lungs.

Angiotensin II also acts on the adrenal glands and releases Aldosterone, which stimulates the epithelial cells in the distal tubule and collecting ducts of the kidneys to increase re-absorption of sodium and water, leading to raised blood volume and raised blood pressure.

This is driven by sodium transport from the lumen into the blood by the Na < sup >+</ sup >/ K < sup >+</ sup > ATPase in the basolateral membrane of the epithelial cells.

CP epithelial cells actively transport sodium, chloride and bicarbonate ions into the ventricles and water follows the resulting osmotic gradient.

In his current work with his brother Prof. Aaron Hanukoglu ( Tel Aviv University, Sackler Medical School and Wolfson Hospital ), he studies the molecular genetics and structure and function of epithelial sodium channels ( ENaC ).

Gilead expanded its move into respiratory therapeutics in 2007 by entering into a licensing agreement with Parion for an epithelial sodium channel inhibitor for the treatment of pulmonary diseases, including cystic fibrosis, chronic obstructive pulmonary disease and bronchiectasis.

By lowering the sodium concentration within the epithelial cells, thiazides increase the activity of the Na < sup >+</ sup >/ Ca < sup > 2 +</ sup >- ATPase on the basolateral membrane to pump more Ca < sup > 2 +</ sup > into the interstitium.

The potassium-sparing diuretics are competitive antagonists that either compete with aldosterone for intracellular cytoplasmic receptor sites, or directly block sodium channels ( specifically epithelial sodium channels ( ENaC ) by amiloride ).

Triamterene directly blocks the epithelial sodium channel ( ENaC ) on the lumen side of the kidney collecting tubule.