* Acetylcholinesterase inhibitors: neostigmine and pyridostigmine can improve muscle function by slowing the natural enzyme cholinesterase that degrades acetylcholine in the motor end plate ; the neurotransmitter is therefore around longer to stimulate its receptor.

Category: Acetylcholinesterase inhibitors

Category: Acetylcholinesterase inhibitors

* Acetylcholinesterase inhibitors ( abbreviated AChEIs )

Category: Acetylcholinesterase inhibitors

* Acetylcholinesterase inhibitors, highly toxic to other animals and humans, acts also as a contact poison

* Acetylcholinesterase inhibitors

Category: Acetylcholinesterase inhibitors

Category: Acetylcholinesterase inhibitors

* Acetylcholinesterase () ( AChE ), also known as RBC cholinesterase, erythrocyte cholinesterase, or ( most formally ) acetylcholine acetylhydrolase, found primarily in the blood and neural synapses.

Acetylcholinesterase exists in multiple molecular forms.

“ Mechanisms of Neuroprotective Effects of Nicotine and Acetylcholinesterase Inhibitors: Role of α4 and α7 Receptors in Neuroprotection ” Journal of Molecular Neuroscience 40. 1-2.

“ Acetylcholinesterase Inhibitors Used in Treatment of Alzheimer ’ s Disease Prevent Glutamate Neurotoxicity via Nicotinic Acetylcholine Receptors and Phospha tidylinositol 3-kinase Cascade ” Neuropharmacology 51. 3.

These include monoclonal antibodies and the new tyrosine kinase inhibitors e. g. imatinib mesylate ( Gleevec or Glivec ), which directly targets a molecular abnormality in certain types of cancer ( chronic myelogenous leukemia, gastrointestinal stromal tumors ).

Since 2008 there have been other drugs ( e. g. kinase inhibitors and anti-VEGF ) available.

Recent scientific studies have shown that cognitive behavioral therapy ( CBT ) and selective serotonin reuptake inhibitors ( SSRIs ; e. g., fluoxetine and paroxetine ) are effective treatment options for hypochondriasis as demonstrated in clinical trials.

GH release in the pituitary is primarily determined by the balance of these two peptides, which in turn is affected by many physiological stimulators ( e. g., exercise, nutrition, sleep ) and inhibitors ( e. g., free fatty acids ) of GH secretion.

Drugs affecting the central nervous system include: hypnotics, anaesthetics, antipsychotics, antidepressants ( including tricyclic antidepressants, monoamine oxidase inhibitors, lithium salts, and selective serotonin reuptake inhibitors ( SSRIs )), antiemetics, anticonvulsants / antiepileptics, anxiolytics, barbiturates, movement disorder ( e. g., Parkinson's disease ) drugs, stimulants ( including amphetamines ), benzodiazepines, cyclopyrrolones, dopamine antagonists, antihistamines, cholinergics, anticholinergics, emetics, cannabinoids, and 5-HT ( serotonin ) antagonists.

* As topiramate inhibits carbonic anhydrase, use with other inhibitors of carbonic anhydrase ( e. g. acetazolamide ) increases the risk of kidney stones.

Remissions may be induced with steroids, chemotherapy, proteasome inhibitors ( e. g. bortezomib ), immunomodulatory drugs ( IMiDs ) such as thalidomide or lenalidomide, and stem cell transplants.

A negative multiplication effect can be achieved by transmitting signal inhibitors ( i. e. oppositely charged ions ) along the dendrite in response to the reception of synaptic neurotransmitters.

Some DMARDs ( e. g. the Purine synthesis inhibitors ) are mild chemotherapeutics but use a side-effect of chemotherapy-immunosuppression-as its main therapeutical benefit.

The sulfonamide chemical moiety is also present in other medications that are not antimicrobials, including thiazide diuretics ( including hydrochlorothiazide, metolazone, and indapamide, among others ), loop diuretics ( including furosemide, bumetanide, and torsemide ), sulfonylureas ( including glipizide, glyburide, among others ), and some COX-2 inhibitors ( e. g., celecoxib ), and acetazolamide.

Vasodilator agents ( e. g., ACE inhibitors, α1-antagonists ) can lead to substantial improvements in neuronal blood flow, with corresponding improvements in nerve conduction velocities.

*-coxib for COX-2 inhibitors, a type of anti-inflammatory drugs ( e. g. celecoxib )

*-pril for ACE inhibitors ( e. g. captopril )

*-vastatin for HMG-CoA reductase inhibitors, a group of cholesterol lowering agents ( e. g. simvastatin )

In theory, this selectivity allows celecoxib and other COX-2 inhibitors to reduce inflammation ( and pain ) while minimizing gastrointestinal adverse drug reactions ( e. g. stomach ulcers ) that are common with non-selective NSAIDs.

* Certain medications e. g. isotretinoin, estrogen, hydrochlorothiazide diuretics, beta blockers, protease inhibitors

Protease inhibitors ( PIs ) are a class of drugs used to treat or prevent infection by viruses, including HIV and Hepatitis C. PIs prevent viral replication by inhibiting the activity of proteases, e. g. HIV-1 protease, enzymes used by the viruses to cleave nascent proteins for final assembly of new virions.

Acanthosis nigricans may also be seen with certain medications that lead to elevated insulin levels ( e. g., glucocorticoids, niacin, insulin, oral contraceptives, and protease inhibitors ).

* Proton pump inhibitors, e. g. esomeprazole

Ventricular septum defect in infants is initially treated medically with cardiac glycosides ( e. g., digoxin 10-20mcg / kg per day ), loop diuretics ( e. g., furosemide 1 – 3 mg / kg per day ) and ACE inhibitors ( e. g., captopril 0. 5 – 2 mg / kg per day ).

The rust inhibitors in the fluid are used up after one year, and you don't want to risk the rust that two years' use could mean.

Selective inhibitors can distinguish the two activities.

Palmer amaranth is among the " top five most troublesome weeds " in the southeast of the United States and has already evolved resistances to dinitroanilines and acetolactate synthase inhibitors.

* Beta-lactamase inhibitors, such as clavulanic acid and sulbactam.

After widespread adoption of the COX-2 inhibitors, it was discovered that most of the drugs in this class increased the risk of cardiovascular events by 40 % on average.

Drugs including the monoamine oxidase inhibitors ( MAOIs ), tricyclic antidepressants ( TCAs ), tetracyclic antidepressants ( TeCAs ), selective serotonin reuptake inhibitors ( SSRIs ), and serotonin-norepinephrine reuptake inhibitors ( SNRIs ) are most commonly associated with the term.

Selective serotonin reuptake inhibitors ( SSRIs ) are the class of antidepressants commonly used as the first-line treatment for depression because they have a favorable side effect profile and low toxicity.

Serotonin-norepinephrine reuptake inhibitors ( SNRIs ) are a newer form of antidepressants that work on both norepinephrine and 5-HT.

Changing the balance of serotonin and norepinephrine would help the brain cells send and receive messages, therefore boosting the mood, which also is the main reason why these types of medications are called dual reuptake inhibitors.

When a hypothetical dilemma was given to 24 people and according to the dilemma they had the capability of pushing a stranger in front of a train so they could rescue five people, individuals who had taken selective serotonin reuptake inhibitors were not as likely to support the idea of pushing the person.

Norepinephrine-dopamine reuptake inhibitors inhibit the neuronal reuptake of dopamine and norepinephrine.

Irreversible monoamine oxidase inhibitors ( MAOIs ) may be used if other antidepressant medications are ineffective.

Some viruses express caspase inhibitors that inhibit caspase activity and an example is the CrmA protein of cowpox viruses.

Furthermore, many viruses express p53 inhibitors that can bind p53 and inhibit its transcriptional transactivation activity.

The characterization of the caspases allowed the development of caspase inhibitors which can be used to determine whether a cellular process involves active caspases.

Using these inhibitors it was discovered that cells can die while displaying a morphology similar to apoptosis without caspase activation.

The treatment of ulcers resulting from excessive acidity may require H2-receptor antagonists or proton pump inhibitors, and eradication of H. pylori.

Originally synthesized from compounds found in pit viper venom, ACE inhibitors inhibit angiotensin-converting enzyme ( a component of the blood pressure-regulating renin-angiotensin system ), thereby decreasing the tension of blood vessels and blood volume, thus lowering blood pressure.

Frequently prescribed ACE inhibitors include Zapril, perindopril, captopril, enalapril, lisinopril, and ramipril.

ACE inhibitors are used primarily to treat hypertension, although they may also be prescribed for cardiac failure, diabetic nephropathy, chronic renal failure, renal involvement in systemic sclerosis ( scleroderma renal crisis ), left ventricular systolic dysfunction, and acute myocardial infarction.

Angiotensin-converting enzyme inhibitors reduce the activity of the renin-angiotensin-aldosterone system.

ACE inhibitors block the conversion of angiotensin I to angiotensin II.