* Vitamin deficiencies at extremely high doses to the complete diet

Vitamin deficiencies and chronic infections may also occur at any age ; they usually cause other symptoms before dementia occurs, but occasionally mimic degenerative dementia.

Category: Vitamin deficiencies

However, many say that the risk of skin cancer from exposure to the sun outweighs the risk of cancers associated with deficiencies in Vitamin D. In particular, they contribute to a predisposition for hypovitaminosis D, which can lead to rickets or osteoporosis and may increase the risk of seizures in infants born to affected mothers.

Category: Vitamin deficiencies

Vitamin C deficiencies may lead to scurvy which impairs wound healing, which can contribute to ulcer formation.

Category: Vitamin deficiencies

Category: Vitamin deficiencies

* Splitting and fraying are associated with psoriasis and deficiencies of folic acid, protein and Vitamin C.

Category: Vitamin deficiencies

Vitamin deficiencies can lead to trouble shedding skin, lethargy and weight loss ; a calcium deficiency can lead to Metabolic Bone Disease, which can be fatal.

Category: Vitamin deficiencies

Vegans who do not eat foods or pills fortified with synthetic vitamin D and with little exposure to the sun's ultraviolet radiation ( e. g., those who don't expose their extremities for at least 15 – 30 minutes per day or those living at latitudes close to the poles ) are vulnerable to Vitamin D deficiencies.

Vitamin deficiencies can lead to wide ranging clinical abnormalities that reflect the diversity of their metabolic roles.

* Vitamin B < sub > 12 </ sub > and folic acid deficiencies: anaemia

* Vitamin D and calcium deficiencies: spasm, bone pain, numbness and tingling sensation

Category: Vitamin deficiencies

New research is finding a link between Vitamin D deficiencies and cognitive impairment which includes memory loss and a foggy brain.

Anemia and nutritional deficiencies ( such as a deficiency in niacin, riboflavin, iron, or Vitamin E ) must be treated, often by dietary changes or other supplements.

Category: Vitamin deficiencies

* Vitamin deficiencies such as Vitamin B12 ( cobalamin )

Vitamin B < sub > 12 </ sub > deficiency may cause, among several neurological abnormalities, overlapping cerebellar and sensory ataxia.

Vitamin C is especially prone to oxidation during cooking and may be completely destroyed by protracted cooking.

Calcium, Vitamin D, and phosphorus are inter-related ; the consumption of each may affect the absorption of the others.

Rarely, Komondors may have an autosomal recessive gene which prevents absorption of Vitamin B12.

Some are produced by the human body with adequate precursors ( glutathione, Vitamin C ), and those the body cannot produce may only be obtained in the diet via direct sources ( Vitamin C in humans, Vitamin A, Vitamin K ) or produced by the body from other compounds ( Beta-carotene converted to Vitamin A by the body, Vitamin D synthesized from cholesterol by sunlight ).

Because the body uses gastric acid to release B < sub > 12 </ sub > from food particles, decreased vitamin B < sub > 12 </ sub > absorption may occur with long-term use of proton-pump inhibitors and may lead to Vitamin B12 deficiency.

Vitamin P may refer to:

Vitamin E does not decrease mortality in adults, even at large doses, and may slightly increase it.

Vitamin E may help prevent or delay coronary heart disease by limiting the oxidation of LDL-cholesterol.

Vitamin E also may help prevent the formation of blood clots, which could lead to a heart attack.

One study suggested that Vitamin E ( as alpha-tocopherol only ) supplementation may increase the risk for heart failure.

* Vitamin A deficiency may also result due to infection.

Vitamin E causes contact dermatitis in up to 33 % of users and in some cases it may worsen scar appearance.

Baking soda may react with acids in food, including Vitamin C ( L-ascorbic acid ).

Deficiency of Vitamin K may also contribute to bleeding disorders because clotting factor maturation depends on Vitamin K.

Vitamin deficiency is a common result of starvation, often leading to anemia, beriberi, pellagra, and scurvy.

Use of green tea in conjunction with anticoagulants may result in reduced effectiveness ; there is a correlation between the quantity of tea consumed and the method of production may affect the amount of Vitamin K.

When all fifty studies are pooled, one would say no effect of Vitamin X was found, because the positive result was not more frequent than chance, i. e. it was not statistically significant.

As a result, Rumford's soup was often supplemented by corn or herring to supply Vitamin C and Vitamin D.

Scurvy is a disease now known to be caused by a deficiency of Vitamin C, but in Lind's day, the concept of vitamins was unknown.

In renal disease, more active forms of Vitamin D such as cholecalciferol or ( 1, 25-dihydroxycholecalciferol or calcitriol which is the main biologically active form of vitamin D ) is used, as the kidney cannot adequately generate calcitriol from calcidiol ( 25-hydroxycholecalciferol ) which is the storage form of vitamin D. In vitamin D assays, vitamin D < sub > 2 </ sub > ( ergocalitrol ) is not accurately measured, therefore vitamin D < sub > 3 </ sub > ( cholecalciferol ) is recommended for supplementation.

In 1934, a Hungarian physician, Paul György discovered a substance that was able to cure a skin disease in rats ( dermititis acrodynia ), this substance he named vitamin B < sub > 6 </ sub >.< ref > Paul György ( 1934 ) " Vitamin B < sub > 2 </ sub > and the pellagra-like dermatitis in rats ," Nature, vol.

Vitamin K deficiency from other causes ( e. g., in malabsorption ) or impaired vitamin K metabolism in disease ( e. g., in hepatic failure ) lead to the formation of PIVKAs ( proteins formed in vitamin K absence ) which are partially or totally non-gamma carboxylated, affecting the coagulation factors ' ability to bind to phospholipid.

The following year, Sir Frederick Hopkins postulated that some foods contained " accessory factors "— in addition to proteins, carbohydrates, fats, and salt — that were necessary for the functions of the human body .< ref > Christiaan Eijkman, Beriberi and Vitamin B < sub > 1 </ sub >, Official Web Site of the Nobel Foundation </ ref > In 1901, Gerrit Grijns ( May 28, 1865 – November 11, 1944 ), a Dutch physician and assistant to Christiaan Eijkman in the Netherlands correctly interpreted the disease as a deficiency syndrome, and between 1910 and 1913, Dr. Edward Bright Vedder established that an extract of rice bran is a treatment for beriberi.

One of his earliest contributions while at the Royal Infirmary, in 1877, was in orthopaedics, by means of the development of the first bone grafts, but also in knee surgery using a special instrument ( Macewen's osteotome ) both techniques becoming key treatments for the highly prevalent disease of rickets ( caused by a lack of Vitamin D ).

* Humans cannot biosynthesize Vitamin C, and so risk scurvy, Vitamin C deficiency disease, if dietary intake of the vitamin is insufficient.

It was a belief he maintained even as he battled heart disease, which he fought both by taking massive doses of Vitamin C and, according to him, by training himself to laugh.

Another newfound use of Pharmacogenetics involves the use of Vitamin E. The Technion Israel Institute of Technology observed that vitamin E can be used to in certain genotypes to lower the risk of cardiovascular disease in patients with diabetes, but in the same patients with another genotype, vitamin E can raise the risk of cardiovascular disease.

Vitamin E hides the effects of WHS, but does not slow the disease.

* Vitamin E: Vitamin E has had varying responses as an antioxidant depending on the neurodegenerative disease that it is being treated.

Vitamin E ineffective for neuroprotection in Parkinson's disease.