[permalink] [id link]
Mutations are considered the driving force of evolution, where less favorable ( or deleterious ) mutations are removed from the gene pool by natural selection, while more favorable ( or beneficial ) ones tend to accumulate.
from
Wikipedia
Some Related Sentences
Mutations and are
Mutations in two regulatory genes of white grapes turn off production of anthocyanins which are responsible for the color of purple grapes.
Mutations are changes in the DNA sequence of a cell's genome and are caused by radiation, viruses, transposons and mutagenic chemicals, as well as errors that occur during meiosis or DNA replication.
Mutations are caused by radiation, viruses, transposons and mutagenic chemicals, as well as errors that occur during meiosis or DNA replication.
Mutations in transpeptidases that lead to reduced interactions with an antibiotic are a significant source of emerging antibiotic resistance.
Mutations are an important force driving evolution.
Mutations that affect insulin-like signaling in worms, flies, and the growth hormone / IGF1 axis in mice are associated with extended lifespan.
Mutations are permanent, transmissible changes to the genetic material ( usually DNA or RNA ) of a cell.
Mutations leading to the loss of function of a gene are much more common than mutations that produce a new, fully functional gene.
Mutations in the tafazzin gene ( TAZ, also called G4. 5 ) are closely associated with
Mutations periodically occur within the Y chromosome and these mutations are passed on to males in subsequent generations.
Mutations of the genes VHL, RET, NF1 ( Gene 17 Neurofibromatosis type 1 ), SDHB and SDHD are all known to cause familial pheochromocytoma / extra-adrenal paraganglioma.
Mutations are characterized as either β < sup > o </ sup > or β thalassemia major if they prevent any formation of β chains, the most severe form of β thalassemia.
Mutations are the ultimate source of genetic variation because they alter the order of bases in the nucleotides of DNA.
Mutations are likely to be rare and most mutations are neutral or deleterious, but in some instances the new alleles can be favored by natural selection.
Mutations in the Ras family of proto-oncogenes ( comprising H-Ras, N-Ras and K-Ras ) are very common, being found in 20 % to 30 % of all human tumours.
Mutations in this gene are rare.
Mutations affecting repressor are said to be recessive to wild type ( and that wild type is dominant ), and this is explained by the fact that repressor is a small protein which can diffuse in the cell.
Mutations usually sound at pitches in the harmonic series of the fundamental and, except where they are derived from unit ranks, are always tuned pure.
Mutations within the desmosome are the main cause of Arrhythmogenic right ventricular cardiomyopathy ( ARVC ).
Mutations of the WT1 gene on chromosome 11 p 13 are observed in approximately 20 % of Wilms ' tumors .< ref > At least half of the Wilms ' tumors with mutations in WT1 also carry mutations in CTNNB1, the gene encoding the proto-oncogene beta-catenin.
Mutations and less
Mutations in the dystrophin gene that lead to the production of less defective, but still only partially functional dystrophin protein, result in a display of a much milder dystrophic phenotype in affected patients, resulting in the disease known as Becker's muscular dystrophy ( BMD ).
Mutations and favorable
Mutations that make organisms more adapted to their environment increase in the population through natural selection as organisms with favorable mutations have more offspring.
Mutations and deleterious
Suddenly, evolutionary theorists could answer the charge that spontaneous random mutations should result overwhelmingly in deleterious changes to a fragile, monolithic genome: Mutations in homeobox regulation could safely — yet dramatically — alter morphology at a high level, without damaging coding for specific organs or tissues.
Mutations and mutations
Mutations in the surface proteins allow the virus to elude some host immunity, and the numbers and locations of these mutations that confer the greatest amount of immune escape has been an important topic of study for over a decade.
Mutations in receptors that result in increased constitutive activity underlie some inherited diseases, such as precocious puberty ( due to mutations in luteinizing hormone receptors ) and hyperthyroidism ( due to mutations in thyroid-stimulating hormone receptors ).
Mutations in the SMPD1 gene cause Niemann – Pick disease types A and B, and mutations in NPC1 and NPC2 cause Niemann – Pick disease, type C ( NPC ).
Mutations tend to fall into two categories, having either a very bad effect or very little effect: few mutations fall somewhere in between.
Mutations in the genes for these keratins are associated with epidermolysis bullosa simplex and Dermatopathia pigmentosa reticularis, both of which are autosomal dominant mutations.
Mutations that are not germline are somatic mutations, which are also called acquired mutations.
Mutations in the same gene also cause enlarged vestibular aqueduct syndrome ( EVA or EVAS ), another congenital cause of deafness ; specific mutations are more likely to cause EVAS, while others are more linked with Pendred syndrome.
Mutations of SDHB play an important role in familial adrenal pheochromocytoma and extra-adrenal paraganglioma ( of abdomen and thorax ), although there is considerable overlap in the types of tumors associated with SDHB and SDHD gene mutations.
Mutations in the SLC26A2 gene cause diastrophic dysplasia, and it is one of a spectrum of skeletal disorders caused by mutations in the SLC26A2 gene.
Mutations in DNA that lead to cancer ( only certain mutations can lead to cancer and the majority of potential mutations will have no bearing ) disrupt these orderly processes by disrupting the programming regulating the processes.
Mutations and from
Mutations in the androgen receptor gene can cause problems with any of the steps involved in androgenization, from the synthesis of the androgen receptor protein itself, through the transcriptional ability of the dimerized, androgen-AR complex.
Beck paid tribute to the group with his single " Tropicália " from the album Mutations.
Mutations in captivity have emerged in various colors, some quite different from those observed in nature.
Mutations that prevent the formation of MinE rings result in MinCD polymers ' extending well beyond the polar zones, preventing division from occurring.
Mutations in the PCCA or PCCB genes disrupt the function of the enzyme, preventing these acids from being metabolized.
*" Mutations ", a song by Salt the Wound from their 2009 album, Ares
* Mutations ( half live and half studio remixes from first album, 1994 )
* Beck's " High Five ( Rock the Catskills )" on the album Odelay ( 1996 ), starting at 2: 10 and finishing at 2: 30 ; and on " Tropicalia " from the album Mutations ( 1998 ) played by Smokey Hormel
Mutations in the SLC26A2 gene alter the structure of developing cartilage, preventing bones from forming properly and resulting in the skeletal problems characteristic of diastrophic dysplasia.
Mutations in the SLC26A2 gene disrupt the structure of developing cartilage, preventing bones from forming properly and resulting in the skeletal problems characteristic of atelosteogenesis, type 2.
Mutations in the KCNJ2 gene alter the usual structure and function of potassium channels or prevent the channels from being inserted correctly into the cell membrane.
Mutations in the ASS gene reduce the activity of the enzyme, which disrupts the urea cycle and prevents the body from processing nitrogen effectively.
Mutations in the HLCS gene reduce the activity of holocarboxylase synthetase, preventing cells from using biotin effectively and disrupting many cellular functions.
Indels can also be contrasted with Tandem Base Mutations ( TBM ), which may result from fundamentally different mechanisms.
Mutations in the COL2A1 gene interfere with the assembly of type II collagen molecules, which prevents bones from developing properly.
Mutations in the COL2A1 gene interfere with the assembly of type II collagen molecules, which prevents bones and other connective tissues from developing properly.
Mutations in the COL2A1 gene that cause Kniest dysplasia interfere with the assembly of type II collagen molecules, which prevents bones from developing properly and causes the signs and symptoms of the disorder.
Mutations in the COL2A1 gene interfere with the assembly of type II collagen molecules, which prevents bones from developing properly and causes the signs and symptoms of this condition.
Mutations in the COL2A1 gene interfere with the assembly of type II collagen molecules, which prevents bones from developing properly and causes the signs and symptoms of this condition.
0.259 seconds.