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Mutations are likely to be rare and most mutations are neutral or deleterious, but in some instances the new alleles can be favored by natural selection.
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Mutations and are
Mutations in two regulatory genes of white grapes turn off production of anthocyanins which are responsible for the color of purple grapes.
Mutations are changes in the DNA sequence of a cell's genome and are caused by radiation, viruses, transposons and mutagenic chemicals, as well as errors that occur during meiosis or DNA replication.
Mutations are caused by radiation, viruses, transposons and mutagenic chemicals, as well as errors that occur during meiosis or DNA replication.
Mutations in transpeptidases that lead to reduced interactions with an antibiotic are a significant source of emerging antibiotic resistance.
Mutations are an important force driving evolution.
Mutations that affect insulin-like signaling in worms, flies, and the growth hormone / IGF1 axis in mice are associated with extended lifespan.
Mutations are permanent, transmissible changes to the genetic material ( usually DNA or RNA ) of a cell.
Mutations are considered the driving force of evolution, where less favorable ( or deleterious ) mutations are removed from the gene pool by natural selection, while more favorable ( or beneficial ) ones tend to accumulate.
Mutations leading to the loss of function of a gene are much more common than mutations that produce a new, fully functional gene.
Mutations in the tafazzin gene ( TAZ, also called G4. 5 ) are closely associated with
Mutations periodically occur within the Y chromosome and these mutations are passed on to males in subsequent generations.
Mutations of the genes VHL, RET, NF1 ( Gene 17 Neurofibromatosis type 1 ), SDHB and SDHD are all known to cause familial pheochromocytoma / extra-adrenal paraganglioma.
Mutations are characterized as either β < sup > o </ sup > or β thalassemia major if they prevent any formation of β chains, the most severe form of β thalassemia.
Mutations are the ultimate source of genetic variation because they alter the order of bases in the nucleotides of DNA.
Mutations in the Ras family of proto-oncogenes ( comprising H-Ras, N-Ras and K-Ras ) are very common, being found in 20 % to 30 % of all human tumours.
Mutations in this gene are rare.
Mutations affecting repressor are said to be recessive to wild type ( and that wild type is dominant ), and this is explained by the fact that repressor is a small protein which can diffuse in the cell.
Mutations usually sound at pitches in the harmonic series of the fundamental and, except where they are derived from unit ranks, are always tuned pure.
Mutations within the desmosome are the main cause of Arrhythmogenic right ventricular cardiomyopathy ( ARVC ).
Mutations of the WT1 gene on chromosome 11 p 13 are observed in approximately 20 % of Wilms ' tumors .< ref > At least half of the Wilms ' tumors with mutations in WT1 also carry mutations in CTNNB1, the gene encoding the proto-oncogene beta-catenin.
Mutations and likely
Mutations in the same gene also cause enlarged vestibular aqueduct syndrome ( EVA or EVAS ), another congenital cause of deafness ; specific mutations are more likely to cause EVAS, while others are more linked with Pendred syndrome.
Mutations and be
One deck represents random Alpha Mutations, which can be drawn to gain temporary powers, and the other contains various Omega Tech, powerful technological devices that could possibly backfire on those that use them.
Mutations can be detected in 90 %.
Mutations can be caused by copying errors in the genetic material during cell division and by exposure to radiation, chemicals, or viruses, or can occur deliberately under cellular control during the processes such as meiosis or hypermutation.
Mutations leading to carcinoma may be accelerated by genetic or environmental factors and other risk factors already described.
Mutations or buffer conditions known to improve aggregation strongly increase the population of the beta conformer, thus suggesting this could be a conformation related to pathogenetic aggregation.
Mutations in some homeotic genes can often be lethal and the cycle of life will end at embryogenesis.
Mutations within the dystrophin gene can either be inherited or occur spontaneously during germline transmission.
Mutations in the Ras / mitogen activated protein kinase signaling pathways are known to be responsible for ~ 70 % of NS cases.
Mutations can be inherited or can arise de novo
Mutations in the MUTYH gene are inherited in an autosomal recessive pattern, which means two copies of the gene must be altered for a person to be affected by the disorder.
Mutations in this gene could be involved in the malignant progression of colon cancer.
Mutations in such genes can be used to classify the malignancy of a tumor.
Mutations in the MTHFR gene could be one of the factors leading to increased risk of developing schizophrenia.
Mutations inherited through the chromosomes can be autosomal dominant or recessive and can also be sex-linked dominant or recessive.
Mutations that occur early on in development will affect a greater number of cells and can result in an individual that can be identified as a mosaic strictly based on phenotype.
Mutations on the other hand can also be part of a survival strategy.
Indels can also be contrasted with Tandem Base Mutations ( TBM ), which may result from fundamentally different mechanisms.
Mutations induced by EMS can then be studied in genetic screens or other assays.
Mutations in G proteins associated with G protein-gated ion channels have been shown to be involved in diseases such as epilepsy, muscular diseases, neurological diseases, and chronic pain, among others.
In humans, GABA < sub > A </ sub >- ρ receptor subunits ρ1 and ρ2 are encoded by the and genes which are found on chromosome 6 whereas the gene for ρ3 is found on chromosome 3 Mutations in the ρ1 or ρ2 genes may be responsible for some cases of autosomal recessive retinitis pigmentosa.
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